Detailed gene information

Genes [306]




STM, SHL, WAM1
SHOOT MERISTEMLESS, SHOOTLESS, WALDMEISTER 1
TAIR: AT1G62360
UNIPROT: Q38874
NOWOMICS: View List

Publications

Clark S E et al., 1996, Development
Long J A et al., 1996, Nature
Endrizzi K et al., 1996, Plant J.
Rupp H M et al., 1999, Plant J.
Aida M et al., 1999, Development
Lenhard M et al., 2002, Development
Bhatt A M et al., 2004, Gene
Uchida N et al., 2007, Proc. Natl. Acad. Sci. U.S.A.
Smith H M S et al., 2011, J. Exp. Bot.
Scofield S et al., 2013, Plant J.
Liebsch D et al., 2014, Development
Aguilar-Martínez J A et al., 2015, Plant Physiol.

Appears in the following schemes


Protein function

Encodes a class I knotted-like homeodomain protein invovled in the control of shoot apical meristem functions.

Phenotype

Single mutant:
stm single mutant lacks an embryonic shoot meristem. They also display different shoot apical meristem defects throughout their life cycle. [Clark et al., 1996]

Overexpressor:
The overexpression of STM leads to the development of ectopic meristems and leaf developmental defects. [Lenhard et al., 2002][Gallois et al., 2002]

Function:
Required for shoot apical meristem (SAM) formation during embryogenesis. Negatively regulates ASYMMETRIC LEAVES1 (AS1) and ASYMMETRIC LEAVES2 (AS2 or LBD6). Probably binds to the DNA sequence 5 -TGAC-3 . [Data from UniProt]

Regulators, targets and interactors

Downstream actors

causality
SOC1, AGL20 -- [López-González et al., 2014] -- [View TAIR record]



Upstream actors


Protein-protein interactions


STM, SHL, WAM1 interaction network

Downstream and upstream flowering-related genes

Physical interactions with other flowering-related proteins