Protein function
Encodes a transcription repressor participating in the polycomb group (PcG) protein complex-mediated silencing of flower homeotic genes.
Phenotype
Single mutant: emf1 single mutant is extremely early flowering under both SD and LD conditions.
[Sanchez et al., 2009][Yang et al., 1995] Remarks: EMF1 is a subunit of a Polycomb repressive complex, which is involved in the repression of
FT expression, thus preventing photoperiod-independent flowering. EMF1 also regulates
AG,
AP3,
PI,
SPLs, and
MIR172 expression.
[Wang et al., 2014] Overexpressor: Overexpression of sense
EMF1 transcripts does not lead to any flowering-time phenotype.
[Aubert et al., 2001] RNA interference: RNAi downregulation of
EMF1 leads to an early flowering phenotype under both SD and LD conditions.
[Aubert et al., 2001][Wang et al., 2014] Function:Transcription repressor that regulates phase transition during shoot, flower and seeds development. Controls leaves development, shoot architecture and flowering by delaying both the vegetative to reproductive transition and flower initiation. Participates in polycomb group (PcG) protein complex-mediated (including EMF2) silencing of the flower homeotic genes AGAMOUS (AG), PISTILLATA (PI), and APETALA3 (AP3), as well as of some regulatory genes such as ABSCISIC ACID INSENSITIVE3 (ABI3), LONG VEGETATIVE PHASE1 (LOV1), and FLOWERING LOCUS C (FLC) during vegetative development. Required for histone methylation or for maintaining a stable histone methylation (e.g. H3K27me3) pattern of repressed target genes. Can bind non specifically DNA (both double- and single-stranded) and RNA.
[Data from UniProt]
Regulators, targets and interactors
Downstream actors
causality AG --
[Calonje et al., 2008] --
[View TAIR record] Upstream actors
Protein-protein interactions
TFL2, LHP1 --
[Wang et al., 2014] --
[View TAIR record]